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With cystic drift, the embryo is also usually absent.

Aplasia can be observed not only in the permanent tissues of the fetus, but also in the afterbirth (placenta, umbilical cord). So, in the placenta in a limited area there may be no thiefblues (fenestrated placenta - placenta fenestrata, placenta fenestrata). Rarely, there is a complete absence of Lasix cord (achordia), always combined with fetal malformations that are incompatible with life; more often there is aplasia of one of the umbilical arteries, which can be combined with congenital malformations of the fetus.

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The effect on the body of aplasia of an organ, tissue or body part is different. Some types of aplasia (anencephaly, amyelia, acardius - acardius and bilateral aplasia of the kidneys) are not compatible with life. Other types can cause severe dysfunction and lead to death at various ages without surgical correction (eg diaphragmatic hernia, Hirschsprung's disease). Ayaplasi limb parts are compatible with life. Aplasia of one of the paired organs, for example, unilateral aplasia of the kidney, is sometimes completely compensated by vicarious hypertrophy of the other and may not manifest itself clinically throughout life. See also Malformations.

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Aplasia in the Encyclopedic Dictionary: Lasix - (from a - negative prefix and Greek plasis - formation) - a malformation, congenital absence of any part of the body or organ. Aplasia of one of the paired organs (for example, one kidney) may not lead to functional violations. Wed Hypoplasia. The meaning of the word Aplasia according to the dictionary of medical terms: aplasia (aplasia; a- Greek plasis formation, education; syn. agenesis) is the general name for developmental anomalies in which a part of the body, an organ or part of it, a section of any tissue is missing.

The causes of aplasia are external teratogenic factors (physical, chemical, biological) that affect the embryo directly or indirectly through the mother's body.
The same factors, acting on the gametes of parents (or more distant ancestors), can cause chromosomal diseases with aplasia of one or another organ (tissue) or a hereditary congenital defect with aplasia.
Aplasia can be observed in any human organ (brain, spinal cord, heart, lungs, urinary-genital organs, etc.).

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Aplasia of the heart (acardius) is observed only with twin congenital malformations.

Aplasia of one of the paired organs usually causes vicarious hypertrophy of the other. Aplasia of one endocrine gland can cause hypo- or hypertrophy of furosemide glands. There may be aplasia of only part of an organ, for example, the olfactory brain (arynencephaly), the corpus callosum, etc. Focal tissue aplasia can be observed in the skin, most often on the scalp, where a defect is found at the birth of a baby, usually not exceeding 5 cm across. Skin aplasia can be combined with aplasia of deeper tissue, for example, in various forms of dysraphia (non-closure of embryonic fissures).

At cystic drift (see) the germ is also, as a rule, absent.
Aplasia can be observed not only in the permanent tissues of the fetus, but also in the afterbirth (placenta, umbilical cord). So, in the placenta in a limited area, villi may be absent (fenestrated placenta - placenta fenestrata). Rarely, there is a complete absence of the umbilical cord (achordia), always combined with fetal malformations that are incompatible with life; more often there is aplasia of one of the umbilical arteries, which can be combined with congenital malformations of the fetus.
The effect on the body of aplasia of an organ, tissue or body part is different.
Some types of aplasia (anencephaly, amyelia, acardius and bilateral aplasia of the kidneys) are not compatible with life. Other types can cause severe dysfunction and lead to death at various ages without surgical correction (eg diaphragmatic hernia, Hirschsprung's disease). Aplasia of a part of a limb is compatible with life. Aplasia of one of the paired organs, for example, unilateral aplasia of the kidney, is sometimes completely compensated by vicarious hypertrophy of the other and may not manifest itself clinically throughout life.
Bone marrow aplasia: what it is and what symptoms it accompanies.
Aplasia of the bone marrow (aplasia of hematopoiesis) - syndromes of bone marrow failure, which are characterized by suppression of hematopoietic functions. Patients are deficient in all types of blood cells: leukocytes, erythrocytes, and platelets. The root cause of aplasia of hematopoiesis is detected using laboratory methods. Methods of lasix depend on the disease that caused the pathology. In the international classification of diseases (ICD-10), bone marrow aplasia is indicated by the code D61. What is bone marrow aplasia?
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Bone marrow is an organ of the hematopoietic system that contains both stem and mature blood cells. A decrease in the number of all blood cells due to acquired (common) or congenital (rare) bone marrow aplasia is called aplastic anemia. Congenital forms include Fanconi anemia and Diamond-Blackfan syndrome.

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Bone marrow aplasia is a condition in which the hematopoietic function of lasix marrow is sharply suppressed. Annually there are 0.2-0.3 cases per 100,000 people. About 200-300 people in US suffer from bone marrow aplasia. The disease is life-threatening and is reflected in the altered blood picture of patients. The diagnosis can affect even healthy young people.

If hematopoiesis in the bone marrow is impaired, defective blood cells may form.

The disorder can affect different types of cells (erythrocytes, leukocytes, platelets). Symptoms of aplasia of the hematopoietic system occur because the number of cells decreases so much that they cannot perform their function sufficiently.

According to the severity of lasix and thrombocytopenia, 3 degrees are distinguished:

A decrease in the concentration of red blood cells causes weakness, fatigue, shortness of breath and heart palpitations, especially during physical exertion. Patients with anemia often have pale skin. With bone marrow aplasia, the immune system is reduced.

Due to the decrease in the number of white blood cells, susceptibility to infectious diseases increases. Since the body's immune system cannot function optimally with a reduced number of granulocytes, infection can be fatal. Therefore, it is important to consult a doctor immediately in such situations.

With a reduced number of Furosemide, the blood coagulation system is disturbed. As a result, so-called petechiae occur - very small pinpoint bleeding or bruising (hematoma). They can also occur spontaneously, without prior trauma. Even relatively minor bleeding or microtrauma (for example, when visiting a dentist) can be fatal.

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According to the etiology (cause of occurrence), congenital and acquired aplasia of the bonebrain. Anemia Fanconi. Diamond-Blackfan Syndrome. Idiopathic (>70% of cases). Medicinal (10%): non-steroidal anti-inflammatory drugs, chloramphenicol, phenylbutazone, gold, penicillamine, allopurinol, phenytoin. Toxic (10%). Viral (5%): especially parvovirus B19 and Epstein-Barr virus. Because in many situations a risk factor cannot be identified, most cases should be classified as idiopathic, with no known cause. However, bone marrow hypoplasia (or aplasia) can also occur as part of an autoimmune disease such as systemic lupus erythematosus.

It is known that a number of cytotoxic drugs increase the risk of developing hypoplasia in the bone marrow. It should be noted that antimetabolites cause only acute aplasia, while alkylating agents cause chronic aplasia.

Hypoplasia, like aplasia, of the bone marrow can be acute or chronic. The first warning signs may be neutropenia and thrombocytopenia. Sometimes there are clinical signs of anemia: fatigue, a general feeling of weakness, pallor of the skin and mucous membranes. In the chronic form, infections develop in the mouth and neck. Sometimes the tendency to bleed increases.